Emideltide · 25–30 nmol/kg IV · SC Unvalidated

DSIP

Dosage & Dose Escalation Guide

Evidence-based DSIP (emideltide) dosage guide covering historical 25–30 nmol/kg IV sleep studies, nmol/kg to mcg/kg conversion, mixed human efficacy, 100–400 mcg SC community conventions, ~8 min IV half-life limits, PCAC 7–6 vote, and proposed SC Phase 1b protocol.

★★★★★4.5(720 reviews)Not FDA Approved · IV Human Data · SC Unvalidated
  • Emideltide
  • 25–30 nmol/kg IV
  • 100–400 mcg SC Anecdotal
  • No Receptor Confirmed
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  • Identity

    WAGGDASGE · MW ~848.8 g/mol · confirm free base vs emideltide acetate assay basis.

  • Human sleep anchor

    25–30 nmol/kg IV (~21–25 mcg/kg, ~1.5–1.8 mg/70 kg) — small trials, mixed results.

  • Community SC

    100–400 mcg before bed — no controlled human SC sleep trial identified.

How It Works

No confirmed receptor. Hypotheses include indirect opioid-system signaling, GABA/glutamate modulation, neuroendocrine effects, and circadian-state dependence. Name association with delta sleep does not prove reliable stage N3 increase. Short IV plasma half-life does not define SC dosing interval.

Mechanism status

  • No confirmed receptor or gene
  • Kovalzon 2006: “unresolved riddle”
  • Endogenous role uncertain

Historical human IV

  • 25–30 nmol/kg sleep studies
  • Mixed efficacy · small n
  • ~209 people IV exposure total

Community SC

  • 100–400 mcg before bed
  • No SC sleep PK or trial
  • ≠ IV nmol/kg without bridge

Result

Human IV Sleep: Mixed

25–30 nmol/kg: Historical

100–400 mcg SC: Anecdotal

Expected Results Over Time

Updated August 2026

DSIP Dosage: Research Protocols, Human Studies, and Safety

Research note: DSIP has been given to humans, but mostly as 25–30 nmol/kg IV in small 1980s–90s sleep studies with mixed results. Contemporary 100–400 mcg SC bedtime protocols are community conventionsno controlled human SC sleep trial identified. Plasma ~8 min IV half-life (limited data) does not define SC dosing interval. No confirmed receptor or gene.

DSIP (emideltide) is a nine-amino-acid peptide (WAGGDASGE, MW ~848.8 g/mol) named for early delta-sleep association. The name is more certain than the biology — no dedicated receptor, gene, or precursor confirmed.

Historical insomnia research used 25–30 nmol/kg IV (~21–25 mcg/kg, ~1.5–1.8 mg for 70 kg). Some studies reported better sleep efficiency or latency; Bes 1992 and Monti 1987 found weak or limited benefit. Online protocols use 100–400 mcg SC — different route, unknown bioavailability.

FDA July 2026 PCAC voted 7–6 against recommending emideltide for 503A compounding. Proposed next study: SC sentinel escalation 50 → 150 → 300 → 600 mcg with PSG, PK, and cardiovascular monitoring.

DSIP dosage in 30 seconds

QuestionCurrent answer
SequenceWAGGDASGE · emideltide · MW ~848.8 g/mol
Human sleep anchor25–30 nmol/kg IV (~21–25 mcg/kg)
70 kg IV total~1.5–1.8 mg
Community SC range100–400 mcg · anecdotal
Human SC sleep trialNone identified
IV half-life (limited)~8 minutes
Receptor / geneNone confirmed
Proposed SC study50–600 mcg SAD · 150/300 mcg × 14 nights MAD

What is DSIP?

Delta sleep-inducing peptide was isolated from cerebral venous blood of sleeping rabbits and later synthesized as a nonapeptide. Emideltide is the recognized nonproprietary name.

DSIP-like immunoreactivity appears in tissues, but a gene encoding DSIP, clear precursor, and dedicated receptor have not been identified. Defensible description: defined synthetic peptide with historical sleep, neuroendocrine, stress, pain, and withdrawal research — endogenous identity and primary mechanism unresolved.

Identity gate

Confirm DSIP / emideltide vs emideltide acetate — assay basis required

Confirm free base vs acetate and peptide-content assay

Trp-Ala-Gly-Gly-Asp-Ala-Ser-Gly-Glu · MW ~848.8 g/mol · CAS 62568-57-4. Free-base and acetate differ in counterion mass, solubility, and assay basis.

Why DSIP is scientifically controversial

Evidence problems

No receptor · small IV trials · mixed sleep outcomes · SC PK unknown

Confirmed receptor
None
Confirmed gene/precursor
None identified
Historical trial size
Mostly 6–18 participants
Sleep outcome consistency
Mixed — weak independent replication
Route of human evidence
Predominantly IV — not SC/nasal
SC human PK
Not characterized
Controlled SC sleep trial
None identified

The name “delta sleep-inducing peptide” should not be treated as proof of reliable stage N3 increase in humans.

Research and compounding status

No FDA-approved indication or prescribing dosage. July 2026 PCAC voted narrowly against 503A inclusion for emideltide free base and acetate. FDA emphasized IV-dominated human data, no SC PK/safety/efficacy, chronic-use uncertainty, and injectable peptide quality concerns.

Converting the historical dose

mcg/kg = nmol/kg × 0.8488 (MW 848.8 g/mol). Calculations compare quantities only — IV, SC, and intranasal are not interchangeable without measured exposure.

Historical IV dose

nmol/kg → mcg/kg → total mg (IV research anchor only)

nmol/kg preset

Most repeated sleep studies

mcg/kg

21.2

Total mcg

1485

Total mg (IV)

1.49 mg

Formula: mcg/kg = nmol/kg × 0.8488. IV historical anchor — not interchangeable with fixed SC mcg without measured bioavailability.

Molar to mass dose

nmol/kgmcg/kg~70 kg total~80 kg total
2521.21.49 mg1.70 mg
3025.51.78 mg2.04 mg
3529.72.08 mg2.38 mg
5042.42.97 mg3.40 mg

Human sleep studies

Most replicated amount: 25–30 nmol/kg IV — not fixed 100–400 mcg SC. Better-controlled independent work (Bes 1992, Monti 1987) was less persuasive than early network studies.

Outcomes varied among stage 2, slow-wave sleep, REM, total sleep time, awakenings, and next-day function — not uniform “deep sleep increase.”

Human sleep literature

25–30 nmol/kg IV · mixed efficacy · small n

StudynDoseFindingLimit
Schneider-Helmert 1981625 nmol/kg IV↑ sleep time in observation periodTiny pilot
Schneider-Helmert 19861830 nmol/kg IVImproved latency, TST, efficiencyNo adequate concurrent control
Monti 1987625 nmol/kg IVSmall stage-2 changes; weak clinical benefitBaseline differences
Bes 19921625 nmol/kg IVNo significant overall sleep improvementIndependent negative-leaning result
Hruz 2001Small5 mcg/kg IN↑ P300 amplitudeNot insomnia trial

Other human research

Withdrawal (Dick, Backmund): 25–35 nmol/kg IV intensive regimens — open-label, hypotension cases, not home protocols. ACTH/cortisol: mixed — Bjartell ↓ ACTH; Späth-Schwalbe no CRH/meal effect. Anesthesia (Pomfrett 2009): 25–100 nmol/kg IV — ↑ HR, ↓ HRV under isoflurane.

FDA identified 25–150 nmol/kg IV in ~209 people over 1–15 days — exposure total, not efficacy proof.

Commonly reported research protocols

Reported protocols

100–400 mcg SC community conventions — no controlled SC sleep trial

Dose
150–300 mcg
Frequency
Once before bed
Route
SC
Duration
2–4 weeks
Evidence basis
Repeated online/clinic convention

Anecdotal versus clinically studied dosing

Evidence split

Historical IV nmol/kg vs contemporary fixed SC mcg

Historical human research (mostly IV)

25–30 nmol/kg · small trials · mixed results

Typical dose
25–30 nmol/kg (~21–25 mcg/kg)
Route
Intravenous infusion/bolus
70 kg total
~1.5–1.8 mg IV
Duration
Often 1–7 treatment days
Monitoring
Sleep lab / PSG in some studies
SC sleep trial
None identified

Contemporary community protocols

100–400 mcg SC — route/PK unvalidated

Typical dose
100–300 mcg fixed SC
Route
Subcutaneous; sometimes intranasal
Timing
30–60 min before bed
Frequency
Nightly, 3× weekly, or 5 on/2 off
Duration
2–8 weeks
Evidence
Uncontrolled reports

For 70 kg, 25 nmol/kg IV ≈ 1.49 mg vs 200 mcg SC ≈ one-seventh nominal mass with unknown bioavailability. Neither direction proves the other route safe or effective.

Body weight and escalation

Historical IV used weight-based dosing; contemporary SC uses fixed mcg. No validated weight-adjustment model for SC. Online 100 → 300 mcg weekly escalation schedules are community conventions, not clinical titration.

Animal and preclinical dosage

Rabbit ICV/IV, rat IV/IP, cat SC, rat intranasal stroke model — inconsistent across species, circadian phase, and route. No animal-to-human conversion establishes contemporary SC protocol when human IV data already exist.

Powder, vial, and subcutaneous math

Community SC amounts require a validated peptide-content assay and sterile/endotoxin-controlled product. Calculator below is arithmetic only — does not validate research-vial injection.

Reconstitution math

Vial mg + diluent → mcg/mL → volume for target SC mcg (arithmetic only)

Concentration

2500 mcg/mL

Volume

0.08 mL

Insulin syringe

8 units

Assumes validated peptide-content assay and sterile/endotoxin-controlled product. Does not validate research-vial injection.

Route problems

Oral: not established. SC: no human sleep PK. Intranasal: one P300 report — not insomnia dose. IV at home: infusion-rate sensitivity in early work — not DIY. Bacteriostatic water reconstitution without validated cosolvent is questionable for poorly soluble material.

Complete proposed SC research protocol

Phase 1b: randomized, double-blind, placebo-controlled SC emideltide in insomnia disorder. Part A SAD: 50, 150, 300, 600 mcg SC 90 min before bed with dense PK (5–480 min). Part B: 14-night MAD — placebo, 150 mcg, 300 mcg nightly with PSG nights 1/7/14.

Proposed Phase 1b SC design

SAD 50 → 150 → 300 → 600 mcg · MAD 150/300 mcg × 14 nights + PSG

Part A — Single ascending dose (SC, 90 min before bed)

CohortDoseNote
A150 mcg SCBelow community range
A2150 mcg SCLower community center
A3300 mcg SCUpper-middle community amount
A4600 mcg SCConditional exposure cohort

Part B — Multiple ascending dose (14 nights)

ArmDoseMonitoring
PlaceboPlaceboPSG nights 1, 7, 14 · PK · BP/HR
150 mcg nightly × 14150 mcg nightlyPSG nights 1, 7, 14 · PK · BP/HR
300 mcg nightly × 14300 mcg nightlyPSG nights 1, 7, 14 · PK · BP/HR

Requires GMP SC formulation, GLP tox by intended route, validated LC-MS/MS, and IRB/regulatory approval — not a personal protocol.

Prerequisites: GMP formulation, GLP tox by intended route, validated LC-MS/MS, aggregate/endotoxin control, IRB/regulatory approval.

Safety and side effects

Safety monitoring

IV withdrawal hypotension · no controlled SC incidence table

IV withdrawal studies (FDA review)
Perspiration, headache, nausea, vertigo; hypotension in several cases — confounded by withdrawal state
Anesthesia study
↑ heart rate, ↓ HRV under isoflurane — not bedtime incidence data
SC community use
No controlled human SC adverse-event incidence table
Product quality
Aggregation, endotoxin, impurities — FDA flagged emideltide characterization gaps

How DSIP may work

No single confirmed receptor. Hypotheses: indirect opioid-system signaling (not direct receptor agonist), GABA/glutamate modulation, neuroendocrine effects, circadian-state dependence. “Facilitates sleep when conditions allow” ≠ proven circadian reset.

Claims versus evidence

Claim checker

Common DSIP claims vs the evidence record

DSIP reliably increases deep delta sleep

Unproven

Stage N3/delta effects are inconsistent across studies. Name ≠ proven biology.

Dosage evidence ladder

Dosage evidence ladder

IV human exposure exists · SC sleep dose unvalidated

LevelWhat existsConfidence
FDA-approved doseNoneNone
Human clinical sleep dosing25–30 nmol/kg IV most commonWeak · mixed efficacy
Broader human IV exposure25–150 nmol/kg · ~209 people · 1–15 daysExposure only
Human intranasal5 mcg/kg P300 study onlyNot sleep efficacy
Anecdotal SC100–400 mcg before bedInsufficient
Long-term human SC safetyNone establishedNone

Bottom line

DSIP has a fragmented human history — mostly IV 25–30 nmol/kg in small trials with mixed sleep findings. Mechanism remains unusually uncertain.

The 100–400 mcg SC range answers a real search question but is community convention, not historical trial dose or validated clinical protocol. The useful next step is route-specific SC study with measured exposure, PSG, next-day function, and cardiovascular monitoring.

IV nmol/kg ≠ SC mcg. Short IV half-life ≠ SC schedule. Name ≠ deep-sleep proof.

Frequently asked questions

What is the usual DSIP research dose?

Historical sleep studies used 25–30 nmol/kg IV. Community protocols report 100–400 mcg SC. Only the former has direct human experimental support; neither is an established clinical dosage.

How much is 25 nmol/kg in micrograms?

About 21.2 mcg/kg, or roughly 1.49 mg total for a 70 kg participant.

Is 300 mcg a standard dose?

It is a commonly repeated community amount, not a dose established by a controlled human SC sleep trial.

What is DSIP's half-life?

FDA cited approximately eight minutes after IV exposure. SC and intranasal human half-lives are not established.

Has subcutaneous DSIP been tested in humans for sleep?

FDA's 2026 evaluation did not identify clinical SC sleep information. Established literature is predominantly IV.

Does DSIP increase deep sleep?

Human literature is inconsistent. Some studies reported sleep changes; stage N3/delta effects are not uniform or reliably replicated.

Can DSIP lower cortisol?

Human neuroendocrine findings are mixed. Broad cortisol-lowering claims are overstated.

Is DSIP the same as emideltide acetate?

Same active sequence possible, but acetate and free-base materials can differ in mass basis, counterion, and solubility. Assay basis must be specified.

Does DSIP need to be cycled?

No study establishes a required cycle. Five-on/two-off and two-to-four-week patterns are community conventions.

What would establish a real DSIP dose?

GMP product, validated SC PK, randomized dose-ranging with polysomnography, next-day performance testing, cardiovascular monitoring, and longer follow-up.

References

Latest Research on DSIP

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